Chondroitin has been proven to replenish the soft tissue structure of the joints through oral intake. Clinically, chondroitin can effectively relieve the pain and inflammation of arthritis, and at the same time, it has the effect of slowing down the aging of the joints.

Human clinical trials have shown that oral chondroitin can increase the secretion of joint fluid and inter-articular lubricant - hyaluronic acid (hyaluronic acid) concentration, and at the same time reduce the activity of chondroitinolytic enzymes, slowing down the degenerative phenomenon of bone and joints, clinically there are also direct injections of hyaluronic acid in the joints to slow down the deteriorating condition of degenerative arthritis. Clinically, there are also injections of hyaluronic acid directly into the joints to slow down the deterioration of degenerative arthritis. Taking chondroitin to increase the concentration of hyaluronic acid in the joints is similar to the principle of this medical behavior.

According to several human clinical studies on oral chondroitin sulfide over the past few years, chondroitin has been shown to improve the clinical symptoms of arthritis by about 50% compared to non-steroidal analgesic and anti-inflammatory drugs (NSAIDs), and at least 25% more effective in the overall medical evaluation of arthritis compared to those who did not use chondroitin or glucosamine (see Glucosamine Introduction). In overall medical evaluation, the improvement in clinical symptoms was at least 25% greater than in those not using chondroitin or glucosamine (see Glucosamine), and was comparable in arthritic populations using chondroitin and glucosamine alone (39-40%), although there was a trend for a slight 1% greater improvement in those using chondroitin alone, and the combined use of chondroitin and glucosamine was significantly more effective than either ingredient alone (about 7-20%).

There are also some about chondroitin in the prevention and treatment of atherosclerosis clinical research reports, experimental results show that the daily consumption of 2,000 mg of chondroitin sulfide coronary arteriosclerosis patients, the proportion of acute attacks is only 1.7% (1/60), compared to the control group did not take chondroitin atherosclerosis patients, there are about 27% (16/60) of the rate of acute coronary heart attack, as for chondroitin in the prevention and treatment of cardiovascular disease. As for the role of chondroitin in cardiovascular disease prevention and treatment of the mechanism, there is no very clear results, but the study showed that long-term use of chondroitin sulfide high-fat diet, the rate of increase in blood fat, seems to be lower than the ethnic groups who do not take chondroitin sulfide, and at the same time have the effect of lowering the formation of blood clots.

In addition, chondroitin is also one of the components of the blood vessel wall, is not chondroitin at the same time has to repair blood vessels because of blood fat deposition caused by wall damage, and reduce the wall because of the uneven surface of the thrombus aggregation reaction, these effects of the mechanism of transfer may be the chondroitin to reduce the rate of acute attacks of coronary heart disease, but biochemistry of the medical profession or need further research to confirm the chondroitin in cardiovascular disease However, further research is needed to confirm the real effect of chondroitin in the prevention of cardiovascular disease.